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Lyme borreliosis from infection to autoimmunity reviewed.
2004 SEP 1 - (NewsRx.com & NewsRx.net) -- Lyme borreliosis, from infection to autoimmunity, is reviewed in a recent issue of Clinical Microbiology and Infection.
"Lyme borreliosis in humans is an inflammatory disease affecting multiple organ systems, including the nervous system, cardiovascular system, joints, and muscles. The causative agent, the spirochete Borrelia burgdorferi, is transmitted to the host by a tick bite. The pathogenesis of the disease in its early stages is associated largely with the presence of viable bacteria at the site of inflammation, whereas in the later stages of disease, autoimmune features seem to contribute significantly. In addition, it has been suggested that chronic persistence of B. burgdorferi in affected tissues is of pathogenic relevance," scientists in Germany report.
"Long-term exposure of the host immune system to spirochetes and/or borrelial compounds may induce chronic autoimmune disease," said S. K. Singh and Hermann J. Girschick at the University of Wurzburg. "The study of bacterium-host interactions has revealed a variety of proinflammatory and also immunomodulatory-immunosuppressive features caused by the pathogen. Therapeutic strategies using antibiotics are generally successful, but chronic disease may require immunosuppressive treatment."
"Effective and safe ...
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Source: HighBeam Research, Lyme borreliosis from infection to autoimmunity reviewed.